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CCRN Pneumothorax

CCRN Pneumothorax Online Review

CCRN Pneumothorax Overview

Pneumothorax

A pneumothorax is an abnormal collection of air or gas in the pleural space that causes an uncoupling of the lung from the chest wall. Like pleural effusion (liquid buildup in that space), pneumothorax may interfere with normal breathing. It is often called collapsed lung, although that term may also refer to atelectasis. One or both lungs may be affected. A primary pneumothorax is one that occurs spontaneously without an apparent cause and in the absence of significant lung disease, while a secondary pneumothorax occurs in the presence of existing lung pathology. Pneumothoraces can be caused by physical trauma to the chest (including blast injury), or as a complication of medical or surgical intervention; in this case it is referred to as a traumatic pneumothorax. In a minority of cases of both spontaneous or traumatic pneumothorax, the amount of air in the chest increases markedly when a one-way valve is formed by an area of damaged tissue, leading to a tension pneumothorax. This condition is a medical emergency that can cause steadily worsening oxygen shortage and low blood pressure. Unless reversed by effective treatment, these sequelae can progress and cause death.

Signs and Symptoms

    • Chest pain
    • Shortness of breath
    • Decreased or absent breast sounds on the affected side
    • Agitation
    • Tachycardia

Causes

A pneumothorax can be caused by:
  • Chest injury. Any blunt or penetrating injury to your chest can cause lung collapse. Some injuries may happen during physical assaults or car crashes, while others may inadvertently occur during medical procedures that involve the insertion of a needle into the chest.
  • Lung disease. Damaged lung tissue is more likely to collapse. Lung damage can be caused by many types of underlying diseases, including chronic obstructive pulmonary disease (COPD), cystic fibrosis and pneumonia.
  • Ruptured air blisters. Small air blisters (blebs) can develop on the top of your lung. These blebs sometimes burst — allowing air to leak into the space that surrounds the lungs.
  • Mechanical ventilation. A severe type of pneumothorax can occur in people who need mechanical assistance to breathe. The ventilator can create an imbalance of air pressure within the chest. The lung may collapse completely.

Risk Factors

Risk factors for a pneumothorax include:
  • Sex. In general, men are far more likely to have a pneumothorax than are women.
  • Smoking. The risk increases with the length of time and the number of cigarettes smoked, even without emphysema.
  • Age. The type of pneumothorax caused by ruptured air blisters is most likely to occur in people between 20 and 40 years old, especially if the person is a very tall and underweight.
  • Genetics. Certain types of pneumothorax appear to run in families.
  • Lung disease. Having an underlying lung disease — especially chronic obstructive pulmonary disease (COPD) — makes a collapsed lung more likely.
  • Mechanical ventilation. People who need mechanical ventilation to assist their breathing are at higher risk of pneumothorax.
  • Previous pneumothorax. Anyone who has had one pneumothorax is at increased risk of another, usually within one to two years of the first.

Diagnosis

  • A pneumothorax is diagnosed by a combination of signs and symptoms, chest X-ray, and CT scan of the chest

Treatment

ccrn pneumothorax online review

  • Chest tube insertion on the affected side
  • Possible needle decompression
  • Oxygen
  • Pain medications
  • Monitor VS
  • ICU admission if severely compromised
 

Critical Care Courses

Overview

  • Elite Reviews Offers A Variety Of Online Courses That Will More Than Adequately Help Prepare The Critical Care Nurse To Pass The National Exam.
  • Each Course Includes Continuing Education Credit and Sample Questions.

Continuing Education

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CCRN Free Trial
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How It Works

How It Works

  • First - Purchase The Course By Clicking On The Blue Add To Cart Button - You Will Then Be Prompted To Create A User Account.
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  • Third - You Will Be Prompted To Pay For The Review Using PayPal - After Payment You Will Be Redirected Back To Your Account.
  • Last - Click The Start Button Located Within Your Account To Begin The Program

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PCCN Cardiac Exam Overview

PCCN Cardiac Exam Overview

PCCN Cardiac Exam Overview

The Cardiac portion of the PCCN exam covers the majority of the exam.  The cardiac section will consist of roughly 20 to 25 questions on the PCCN exam.  This is the majority of the exam so it is imperative that you focus a lot of your attention on this area.  This post will give you a brief outline of what to expect from the cardiac portion of PCCN exam.  We will briefly cover the cardiovascular system and some of its major components.  Listed below is a detailed outline of what to expect from the cardiac portion of the PCCN exam followed by a brief overview of the cardiovascular system.

Cardiovascular

  • ACS, Angina, Acute MI
  • Acute Heart Failure/Pulmonary Edema
  • Acute Inflammatory Disease
  • Acute Peripheral Vascular Insufficiency
  • Cardiac Surgery and Trauma
  • Cardiac Tamponade
  • Cardiogenic Shock
  • Cardiomyopathies
  • Hypertensive Crisis
  • Hypovolemic Shock
  • Valvular Defects
  • Ruptured or Dissecting Aneurysms
  • ECG/Pacemakers, Pharmacology

PCCN Cardiovascular Exam 

The circulatory system, also called the cardiovascular or vascular system, is an organ system that permits blood to circulate and transport nutrients, oxygen, carbon dioxide, hormones, and blood cells to and from the cells in the body to provide nourishment and help in fighting diseases, stabilize temperature and pH, and maintain homeostasis.  The study of the blood flow is called hemodynamics.  The study of the properties of the blood flow is called hemorrheology.  Take a look at some of the cardiac practice questions below.

PCCN Cardiac Exam Prep Questions

1) Which of the following rhythm disturbances would be most likely to occur in a patient who has a serum potassium level of 8.1 mEq/L?

A) Second-degree heart block, Mobitz Type II, and asystole B) PAC's and ventricular tachycardia C) Right bundle-branch block D) Paroxysmal atrial tachycardia

2) A 65 y/o patient admitted with CHF develops hypotension, tachycardia, decreasing UO, cool clammy skin, decreasing LOC, and tachypnea. Which of the following would be included in the patient's plan of care?

A) Positive inotropic agents, vasodilators, diuretics B) ACE inhibitors, adenosine, beta-blockers C) Beta-blockers, diuretics, calcium channel blockers D) Negative inotropic agents, digoxin, antidysrhythmics

3) A 57 year old patient is experiencing insomnia and associated agitation. The patient prefers not to take a controlled substance as a sleep aid. In addition to reducing noise and interruptions, which of the following interventions has been shown to be both valuable and feasible in this situation?

A) Massage therapy B) Aromatherapy C) Alternative sedative D) Progressive muscle relaxation

4) A 47 year old patient from the Mediterranean is admitted for unstable angina. The patient reports substernal chest pain 8/10, radiating down his left arm. VS are BP 134/76 mm Hg, HR 104 bpm, RR 22 bpm, Temp 98.4. Which of the following meds should be administered with caution to this patient?

A) Aspirin B) Lopressor C) Nitroglycerin D) Morphine

5) The nurse is caring for a 39 y/o male patient who had a CABG 5 days ago. The patient has become increasingly anxious and irritable over the past 4 to 6 hrs. He sometimes holds his head and says it hurts but is unable to grade the pain on a scale of 1 to 10. He is oriented to person. Which of the following interventions should the nurse perform first?

A) Dipstick urine for specific gravity B) Medicate with oral Tylenol C) Measure capillary glucose levels D) Apply soft limb restraints to wrists

PCCN Cardiac Exam Prep Questions Answer with Rationale

1) Correct Answer - A) Second-degree heart block, Mobitz Type II, and asystole
  • Rationale - Hyperkalemia causes depression in AV conduction and leads to heart block.
2) Correct Answer - A) Positive inotropic agents, vasodilators, diuretics 3) Correct Answer - A) Massage therapy
  • Rationale - Studies have demonstrated that massage, music therapy, and therapeutic touch promote relaxation and comfort to critically ill patients.
4) Correct Answer - A) Aspirin
  • Rationale - Many Mediterranean males have a glucose-6-phosphate dehydrogenase enzyme deficiency. This genetic variation increases the risk for hemolysis if the patient receives aspirin.
5) Correct Answer - C) Measure capillary glucose levels
  • Rationale - The patient is exhibiting anxiousness, irritability, and confusion, all of which could be a sign of hypoglycemia.
 

PCCN National Exam Courses

Overview

  • Elite Reviews Offers A Variety Of Online Courses That Will More Than Adequately Help Prepare The Critical Care Nurse To Pass The National Exam.
  • Each Course Includes Continuing Education Credit and Sample Questions.

Continuing Education

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How The Course Works

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  • Second - After Creating An Account, All 3 Options (90, 120 or 150 Days) Will Be Listed. Select The Option You Desire And Delete The Other Two.
  • Third - You Will Be Prompted To Pay For The Review Using PayPal - After Payment You Will Be Redirected Back To Your Account.
  • Last - Click The Start Button Located Within Your Account To Begin The Program

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NCLEX Chronic Renal Failure

NCLEX Chronic Renal Failure

NCLEX Chronic Renal Failure

Chronic Renal Failure Overview

Chronic renal failure, also known as, chronic renal disease, is a progressive loss in kidney function over a period of months or years.  The symptoms of worsening kidney function are not specific, and might include feeling generally unwell and experiencing a reduced appetite.  Often, chronic kidney disease is diagnosed as a result of screening of people known to be at risk of kidney problems, such as those with high blood pressure or diabetes and those with a blood relative with chronic renal disease.  This disease may also be identified when it leads to one of its recognized complications, such as cardiovascular disease, anemia, pericarditis or renal osteodystrophy. Chronic kidney disease is identified by a blood test for creatinine, which is a breakdown product of muscle metabolism.  Higher levels of creatinine indicate a lower GFR and as a result a decreased capability of the kidneys to excrete waste products.  Creatinine levels may be normal in the early stages of chronic renal disease, and the condition is discovered if urinalysis shows the kidney is allowing the loss of protein or red blood cells into the urine.  To fully investigate the underlying cause of kidney damage, various forms of medical imaging, blood tests, and sometimes a kidney biopsy are employed to find out if a reversible cause for the kidney malfunction is present.

Signs and Symptoms

Signs and symptoms of kidney disease are often nonspecific, meaning they can also be caused by other illnesses.  Because your kidneys are highly adaptable and able to compensate for lost function, signs and symptoms may not appear until irreversible damage has occurred.
  • Nausea and vomiting
  • Loss of appetite
  • Fatigue and weakness
  • Sleep problems
  • Changes in urine output
  • Muscle twitches and cramps
  • Swelling of feet and ankles
  • Chest pain, shortness of breath
  • HTN that's difficult to control

Causes

  • Type 1 or Type 2 Diabetes
  • High blood pressure
  • Glomerulonephritis
  • Interstitial nephritis
  • Polycystic kidney disease
  • Prolonged obstruction of the urinary tract
  • Vesicoureteral reflux
  • Pyelonephritis

Risk Factors

  • Diabetes, HTN
  • Smoking
  • Heart disease
  • Obesity
  • High cholesterol
  • African Americans
  • Family history of kidney disease
  • Age 65 or older

Complications

  • Fluid retention - pulmonary edema
  • Hyperkalemia
  • Cardiac disease
  • Osteoporosis
  • Decreased sex drive or impotence
  • Anemia
  • Decreased immune response
  • Pericarditis

Treatments

Depending on the underlying cause, some types of kidney disease can be treated.  Often, though, chronic kidney disease has no cure.  In general, treatment consists of measures to help control signs and symptoms, reduce complications, and slow the progression of the disease.
  • Antihypertensives
  • Medications to lower cholesterol levels
  • Treat anemia
  • Diuretics to relieve the swelling
  • Vitamin D supplements
  • Low protein diet to minimize waste products in your blood
  • Hemodialysis or Peritoneal dialysis
 

NCLEX National Exam Courses

Overview

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  • Third - You Will Be Prompted To Pay For This Review Using PayPal - After Payment You Will Be Redirected Back To Your Account.
  • Last - Click The Start Button Located Within Your Account To Begin The Course

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CEN Brain Herniation

 CEN Brain Herniation Review

CEN Brain Herniation - Overview

Brain herniation is a potentially deadly side effect of very high pressure within the skull that occurs when a part of the brain is squeezed across structures within the skull. The brain can shift across such structures as the falx cerebri, the tentorium cerebelli, and even through the foramen magnum (the hole in the base of the skull through which the spinal cord connects with the brain). Herniation can be caused by a number of factors that cause a mass effect and increase intracranial pressure (ICP): these include traumatic brain injury, intracranial hemorrhage, or brain tumor. Herniation can also occur in the absence of high ICP when mass lesions such as hematomas occur at the borders of brain compartments. In such cases local pressure is increased at the place where the herniation occurs, but this pressure is not transmitted to the rest of the brain, and therefore does not register as an increase in ICP.[2] Because herniation puts extreme pressure on parts of the brain and thereby cuts off the blood supply to various parts of the brain, it is often fatal. Therefore, extreme measures are taken in hospital settings to prevent the condition by reducing intracranial pressure, or decompressing (draining) a hematoma which is putting local pressure on a part of the brain.

Classification

The tentorium is an extension of the dura mater that separates the cerebellum from the cerebrum. There are two major classes of herniation: supratentorial and infratentorial. Supratentorial herniation is of structures normally above the tentorial notch and infratentorial is of structures normally below it. Supratentorial herniation
  • Uncal (transtentorial)
  • Central
  • Cingulate (subfalcine/transfalcine)
  • Transcalvarial
  • Tectal (posterior)
Infratentorial herniation
  • Upward (upward cerebellar or upward transtentorial)
  • Tonsillar (downward cerebellar)

Signs and Symptoms

  • Abnormal posturing
  • Altered LOC
  • Nausea and vomiting
  • Dilated pupils

Treatment

  • Treatment involves removal of the etiologic mass and decompressive craniectomy
  • Ventriculostomy
  • Mechanical ventilation
  • Monitor ICP 
 

Emergency Room Certification Courses

Overview

  • Elite Reviews Offers A Variety Of Online Courses That Will More Than Adequately Help Prepare The Emergency Nurse To Pass The National Exam.
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Continuing Education

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How It Works

How The Course Works

  • First - Purchase The Course By Clicking On The Blue Add To Cart Button - You Will Then Be Prompted To Create A User Account.
  • Second - After Creating An Account, All 3 Options (90, 120 or 150 Days) Will Be Listed. Select The Option You Desire And Delete The Other Two.
  • Third - You Will Be Prompted To Pay For The Review Using PayPal - After Payment You Will Be Redirected Back To Your Account.
  • Last - Click The Start Button Located Within Your Account To Begin The Program

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CCRN Cardiac Tamponade

CCRN Cardiac Tamponade Review

CCRN Cardiac Tamponade

Cardiac tamponade, also known as pericardial tamponade, is a type of pericardial effusion in which fluid, pus, blood, clots, or gas accumulates in the pericardium (the sac in which the heart is enclosed), resulting in slow or rapid compression of the heart. Cardiac tamponade is pressure on the heart muscle which occurs when the pericardial space fills up with fluid faster than the pericardial sac can stretch. If the amount of fluid increases slowly (such as in hypothyroidism) the pericardial sac can expand to contain a liter or more of fluid prior to tamponade occurring. If the fluid effusion occurs rapidly (as may occur after trauma or myocardial rupture) as little as 100 mL can cause tamponade. Myocardial rupture is a somewhat uncommon cause of pericardial tamponade. It typically happens in the subacute setting after a heart attack, in which the infarcted muscle of the heart thins out and tears. Myocardial rupture is more likely to happen in elderly individuals without any previous cardiac history who suffer from their first heart attack and are not revascularized either with thrombolytic therapy or with percutaneous coronary intervention or with coronary artery bypass graft surgery. One of the most common settings for cardiac tamponade is in the first 24 to 48 hours after heart surgery. After heart surgery, chest tubes are placed to drain blood. These chest tubes, however, are prone to clot formation. When a chest tube becomes occluded or clogged, the blood that should be drained can accumulate around the heart, leading to tamponade. Nurses will frequently clear clots from the tubes, but even with these efforts chest tubes can become clogged.

Signs and Symptoms

  • anxiety and restlessness
  • low blood pressure
  • weakness
  • chest pain radiating to your neck, shoulders, or back
  • trouble breathing or taking deep breaths
  • rapid breathing
  • discomfort that’s relieved by sitting or leaning forward
  • fainting, dizziness, and loss of consciousness

Causes

  • gunshot or stab wounds
  • blunt trauma to the chest from a car or industrial accident
  • accidental perforation after cardiac catheterization, angiography, or insertion of a pacemaker
  • punctures made during placement of a central line, which is a type of catheter that administers fluids or medications
  • cancer that has spread to the pericardial sac, such as breast or lung cancer
  • a ruptured aortic aneurysm
  • pericarditis, an inflammation of the pericardium
  • lupus, an inflammatory disease in which the immune system mistakenly attacks healthy tissues
  • high levels of radiation to the chest
  • hypothyroidism, which increases the risk for heart disease
  • a heart attack
  • kidney failure
  • infections that affect the heart

Complications

  • Heart failure
  • Pulmonary edema
  • Shock
  • Death

Treatment

  • Pericardiocentesis
  • Pericardial Window
  • Oxygen
  • Thoracotomy
  • Chest tubes
 

Critical Care Courses

Overview

  • Elite Reviews Offers A Variety Of Online Courses That Will More Than Adequately Help Prepare The Critical Care Nurse To Pass The National Exam.
  • Each Course Includes Continuing Education Credit and Sample Questions.

Continuing Education

  • Each Of Our Online Courses Has Been Approved Continuing Education Contact Hours by the California Board of Nursing
  • Login To Your Account In Order To Access The Course Completion Certificate Once The Course Is Complete.
CCRN Free Trial
  • FREE Sample Lecture & Prep Questions
  • Available For 24 Hrs After Registration
  • Click Free Trial Link To Get Started - CCRN Free Trial

 

How It Works

How It Works

  • First - Purchase The Course By Clicking On The Blue Add To Cart Button - You Will Then Be Prompted To Create A User Account.
  • Second - After Creating An Account, All 3 Options (90, 120 or 150 Days) Will Be Listed. Select The Option You Desire And Delete The Other Two.
  • Third - You Will Be Prompted To Pay For The Review Using PayPal - After Payment You Will Be Redirected Back To Your Account.
  • Last - Click The Start Button Located Within Your Account To Begin The Program

CCRN Predictor Exam

CCRN Predictor Exam

  • 150 Sample Questions
  • Q & A With Rationales
  • Approved For 5 CEU's
  • 90 Days Availability
  • Cost $75.00

           

CCRN Question Bank

CCRN Question Bank

  • 1250+ Sample Questions
  • Q & A With Rationales
  • Approved For 25 CEU's
  • 90 Days Availability
  • Cost $200.00

           

CCRN Practice Questions

CCRN Practice Questions Bundle

  • 1350+ Sample Questions
  • Q & A With Rationales
  • Approved For 30 CEU's
  • 90 Days Availability
  • Cost $225.00

             

CCRN Review

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  • Option 1
  • Lectures & 1250+ Questions 
  • Approved For 35 CEU's
  • 90 Days Availability
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CCRN Online Review

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  • Option 2
  • Lectures & 2000+ Questions
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CCRN Online Review

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  • Option 3
  • Lectures & 3000+ Questions
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PCCN Pulmonary Exam Overview

PCCN Pulmonary Exam Overview

PCCN Pulmonary Exam Overview

Pathophysiology

The Pulmonary AKA Respiratory system is a biological system consisting of specific organs and structures used for the process of respiration in an organism.  The respiratory system is involved in the intake and exchange of oxygen and carbon dioxide between an organism and the environment. Parts of the Respiratory System As we breathe, oxygen enters the nose or mouth and passes the sinuses, which are hollow spaces in the skull. Sinuses help regulates the temperature and humidity of the air we breathe. The trachea, also called the windpipe, filters the air that is inhaled, according to the American Lung Association.  It branches into the bronchi, which are two tubes that carry air into each lung.  The bronchial tubes are lined with tiny hairs called cilia.  Cilia move back and forth, carrying mucus up and out.  Mucus, a sticky fluid, collects dust, germs and other matter that has invaded the lungs.  We expel mucus when we sneeze, cough, spit, or swallow. The bronchial tubes lead to the lobes of the lungs.  The right lung has three lobes; the left lung has two.  The left lung is smaller to allow room for the heart.  Lobes are filled with small, spongy sacs called alveoli, and this is where the exchange of oxygen and carbon dioxide occurs. The alveolar walls are extremely thin.  These walls are composed of a single layer of tissues called epithelial cells and tiny blood vessels called pulmonary capillaries. Blood passes through the capillaries.  The pulmonary artery carries blood containing carbon dioxide to the air sacs, where the gas moves from the blood to the air.  Oxygenated blood goes to the heart through the pulmonary vein, and the heart pumps it throughout the body. The diaphragm, a dome shaped muscle at the bottom of the lungs, controls breathing and separates the chest cavity from the abdominal cavity.  When a breath is taken, it flattens out and pulls forward, making more space for the lungs.  During exhalation, the diaphragm expands and forces air out.

 

Structures of the Pulmonary System

  • Airways
  • Blood vessels
  • Chest wall
  • Lungs
    • Lobes
    • Segments
    • Lobules
  • Conducting Airways
    • Upper Airways
      • Nasopharynx
      • Oropharynx
    • Larynx
      • Connects upper and lower airwars
    • Lower airways
      • Trachea
      • Bronchi
      • Terminal bronchioles

Pulmonary and Bronchial Circulation

  • Pulmonary circulation has a lower pressure than the systemic circulation
  • One third of pulmonary vessels are filled with blood at any given time
  • Pulmonary artery divides and enters the lung at the hilus
  • Each bronchus and bronchiole has an accompanying artery or arteriole

Chest Wall and Pleura

  • Chest Wall

    • Skin, ribs and intercostal muscles
    • Thoracic cavity
  • Pleura
    • Serous membrane
    • Parietal and visceral layers
    • Pleural space
    • Pleural fluid

Gas Transport

Four Steps

  • Ventilation of the lungs
  • Diffusion of oxygen from the alveoli into the capillary blood
  • Perfusion of systemic capillaries with oxygenated blood
  • Diffusion of oxygen from systemic capillaries into the cells

Tests of Pulmonary Function

  • Spirometry
  • Diffusion capacity
  • Residual volume
  • Functional residual capacity
  • Total lung capacity
  • Arterial blood gas analysis
  • Chest radiographs
 

PCCN National Exam Courses

Overview

  • Elite Reviews Offers A Variety Of Online Courses That Will More Than Adequately Help Prepare The Critical Care Nurse To Pass The National Exam.
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Continuing Education

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PCCN Free Trial
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How It Works

How The Course Works

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PCCN Predictor Exam

PCCN Predictor Exam

  • 125 Prep Questions
  • Q & A With Rationales
  • Approved For 5 CEU's
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PCCN Question Bank

PCCN Question Bank

  • 1250+ Prep Questions
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  • Approved For 25 CEU's
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PCCN Practice Questions

PCCN Practice Questions Bundle

  • 1350+ Prep Questions
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  • Approved For 30 CEU's
  • 90 Days Availability
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PCCN Review

PCCN Review Course 

  • Option 1
  • Lectures & 1250+ Questions
  • Q & A With Rationales
  • Approved For 35 CEU's
  • 90 Days Availability
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PCCN Online Review

PCCN Online Review

  • Option 2
  • Lectures & 2000+ Questions
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PCCN Online Review

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  • Option 3
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NCLEX Placenta Abruption

NCLEX Placenta Abruption

 

NCLEX Placenta Abruption

Placenta Abruption Overview

Placental abruption is a complication of pregnancy, wherein the placental lining has separated from the uterus of the mother prior to delivery.  It is the most common pathological cause of later pregnancy bleeding.  In humans, it refers to the abnormal separation after 20 weeks of gestation and prior to birth.  Placental abruption is a significant contributor to maternal mortality worldwide; early and skilled medical intervention is needed to ensure a good outcome, and this is not available in many parts of the world.  Treatment depends on how serious the abruption is and how far along the woman is in her pregnancy. Placental abruption has effects on both the mother and fetus.  The effects on the mother depend primarily on the severity of the abruption, while the effects on the fetus depend on both its severity and the gestational age at which it occurs.  The heart rate of the fetus can be associated with the severity.

Signs and Symptoms

In the early stages of placental abruption, there may be no symptoms.  When symptoms develop, they tend to develop suddenly.  Common symptoms include sudden onset abdominal pain, contractions that seem continuous and do not stop, pain in the abdomen and back, vaginal bleeding, enlarged uterus disproportionate to the gestational age of the fetus, decreased fetal movement, and decreased fetal heart rate. A placental abruption caused by arterial bleeding at the center of the placenta leads to sudden development of severe symptoms and life threatening conditions including fetal heart rate abnormalities, severe maternal hemorrhage, and DIC.  Those abruptions caused by venous bleeding at the periphery of the placenta develop more slowly and cause small amounts of bleeding, intrauterine growth restriction, and oligohydraminos.

Classic symptoms of placental abruption include

  • Vaginal bleeding
  • Abdominal pain
  • Back pain
  • Uterine tenderness
  • Rapid uterine contractions

Causes

The specific cause of placental abruption is often unknown.  Possible causes include trauma or injury to the abdomen from an auto accident or fall.

Risk Factors

  • Pre-eclampsia
  • Chronic hypertension
  • Short umbilical cord
  • Prolonged rupture of membranes
  • Thrombophilia
  • Multiparity
  • Multiple pregnancy
  • Maternal age: < 20 or > 35 are at greatest risk

Complications

For the mother, placental abruption can lead to
  • Shock due to blood loss
  • Blood clotting problems (DIC)
  • The need for blood transfusions
  • Failure of the kidneys or other organs
For the baby, placental abruption can lead to
  • Deprivation of oxygen and nutrients
  • Premature birth
  • Stillbirth

Treatments

Treatment depends on the amount of blood loss and the status of the fetus.  If the fetus is less than 36 weeks and neither mother or fetus is in any distress, then they may simply be monitored in hospital until a change in condition or fetal maturity whichever comes first. Immediate delivery of the fetus may be indicated if the fetus is mature of if the fetus or mother is in distress.  Blood volume replacement to maintain blood pressure and plasma replacement to maintain fibrinogen levels may be needed.  Vaginal birth is usually preferred over caesarean section unless there is fetal distress.  C-section is contraindicated in cases of DIC.  Excessive bleeding from uterus may necessitate hysterectomy.  The mother may be given Rhogam if she is Rh negative.  

NCLEX National Exam Courses

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NCLEX Predictor Exam

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NCLEX Review Course

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NCLEX Online Review

NCLEX Online Review

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CEN Retinal Detachment

CEN Retinal Detachment Review

CEN Retinal Detachment

Retinal detachment is a disorder of the eye in which the retina detaches from the retinal pigment epithelium. Retinal detachments can be caused by fluid leaking behind the retina through tears, by traction on the retina, or by fluid exuding from the retina. The visual prognosis of a retinal detachment is dependent on the duration of the detachment, whether the macula was detached, and the underlying health of both the retina and circulatory system of the eye. The retina is a thin layer of light sensitive tissue on the back wall of the eye. The optical system of the eye focuses light on the retina much like light is focused on the film or sensor in a camera. The retina translates that focused image into neural impulses and sends them to the brain via the optic nerve. Occasionally, posterior vitreous detachment, injury or trauma to the eye or head may cause a small tear in the retina. The tear allows vitreous fluid to seep through it under the retina, and peel it away like a bubble in wallpaper.

Signs and Symptoms

A rhegmatogenous retinal detachment is commonly preceded by a posterior vitreous detachment which gives rise to these symptoms:
  • flashes of light (photopsia) – very brief in the extreme peripheral (outside of center) part of vision
  • a sudden dramatic increase in the number of floaters
  • a ring of floaters or hairs just to the temporal (skull) side of the central vision
Although most posterior vitreous detachments do not progress to retinal detachments, those that do produce the following symptoms:
  • a dense shadow that starts in the peripheral vision and slowly progresses towards the central vision
  • the impression that a veil or curtain was drawn over the field of vision
  • straight lines (scale, edge of the wall, road, etc.) that suddenly appear curved (positive Amsler grid test)
  • central visual loss
In the event of an appearance of sudden flashes of light or floaters, an eye doctor needs to be consulted immediately.  A shower of floaters or any loss of vision, too, is a medical emergency.

Risk Factors

The following factors increase your risk of retinal detachment:
  • Aging — retinal detachment is more common in people over age 50
  • Previous retinal detachment in one eye
  • A family history of retinal detachment
  • Extreme nearsightedness (myopia)
  • Previous eye surgery, such as cataract removal
  • Previous severe eye injury
  • Previous other eye disease or inflammation

Diagnosis

  • Retinal examination - The doctor may use an instrument with a bright light and a special lens (ophthalmoscope) to examine the back of your eye, including the retina. The ophthalmoscope provides a highly detailed view, allowing the doctor to see any retinal holes, tears or detachments.
  • Ultrasound imaging - this test if bleeding has occurred in the eye, making it difficult to see your retina.

CEN Retinal Detachment - Types

  • Rhegmatogenous retinal detachment – A rhegmatogenous retinal detachment occurs due to a break in the retina (called a retinal tearthat allows fluid to pass from the vitreous space into the subretinal space between the sensory retina and the retinal pigment epithelium. Retinal breaks are divided into three types – holes, tears and dialyses. Holes form due to retinal atrophy especially within an area of lattice degeneration. Tears are due to vitreoretinal traction. Dialyses are very peripheral and circumferential, and may be either tractional or atrophic. The atrophic form most often occurs as idiopathic dialysis of the young.
  • Exudative, serous, or secondary retinal detachment – An exudative retinal detachment occurs due to inflammation, injury or vascular abnormalities that results in fluid accumulating underneath the retina without the presence of a hole, tear, or break. In evaluation of retinal detachment it is critical to exclude exudative detachment as surgery will make the situation worse, not better. Although rare, exudative detachment can be caused by the growth of a tumor on the layers of tissue beneath the retina, namely the choroid. This cancer is called a choroidal melanoma.
  • Tractional retinal detachment – A tractional retinal detachment occurs when fibrous or fibrovascular tissue, caused by an injury, inflammation or neovascularization, pulls the sensory retina from the retinal pigment epithelium.

Treatment

  • Cryopexy and laser photocoagulation
  • Scleral buckle surgery
  • Pneumatic retinopexy
  • Vitrectomy
 

Emergency Room Certification Courses

Overview

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CEN Predictor Exam

CEN Predictor Exam

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CEN Question Bank

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CEN Review Course 

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CEN Online Review

CEN Online Review 

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CCRN Aortic Aneurysm

CCRN Aortic Aneurysm Review

CCRN Aortic Aneurysm Overview

Thoracic Aortic Aneurysm

A thoracic aortic aneurysm is an aortic aneurysm that presents primarily in the thorax.  A thoracic aortic aneurysm is the "ballooning" of the upper aspect of the aorta, above the diaphragm. Untreated or unrecognized they can be fatal due to dissection or "popping" of the aneurysm leading to nearly instant death. Thoracic aneurysms are less common than an abdominal aortic aneurysm.  However, a syphilitic aneurysm is more likely to be a thoracic aortic aneurysm than an abdominal aortic aneurysm. A thoracic aortic aneurysm may also be called thoracic aneurysm and aortic dissection (TAAD) because an aneurysm can lead to a tear in the artery wall (dissection) that can cause life-threatening bleeding. Small and slow-growing thoracic aortic aneurysms may not ever rupture, but large, fast-growing aneurysms may rupture.  Depending on the size and growth rate of your thoracic aortic aneurysm, treatment may vary from watchful waiting to emergency surgery. Ideally, surgery for a thoracic aortic aneurysm can be planned if necessary.

Signs and Symptoms

Thoracic aortic aneurysms often grow slowly and usually without symptoms, making them difficult to detect. Some aneurysms will never rupture. Many start small and stay small, although many expand over time. How quickly an aortic aneurysm may grow is difficult to predict. As a thoracic aortic aneurysm grows, some people may notice:
  • Tenderness or pain in the chest
  • Back pain
  • Hoarseness
  • Cough
  • Shortness of breath

CCRN Aortic Aneurysm

Aneurysms can develop anywhere along the aorta, which runs from your heart through your abdomen. When they occur in the upper part of the aorta, they are called thoracic aortic aneurysms. Aneurysms can occur anywhere in the thoracic aorta, including the ascending aorta near the heart, the aortic arch in the curve of the thoracic aorta and the descending aorta in the lower part of the thoracic aorta. Aneurysms that form in the lower part of your aorta — called abdominal aortic aneurysms — are more common than thoracic aortic aneurysms. An aneurysm can also occur in between the upper and lower parts of your aorta. This type of aneurysm is called a thoracoabdominal aneurysm.

Causes

Factors that can contribute to an aneurysm's development include:
  • Hardening of the arteries (atherosclerosis). As plaque builds up on your artery walls, they become less flexible, and the additional pressure can cause them to weaken and bulge. High blood pressure and high cholesterol are risk factors for hardening of the arteries. This is more common in older people.
  • Genetic conditions. Younger people with an ascending aortic aneurysm often have a genetic cause. People who are born with Marfan syndrome, a genetic condition that affects the connective tissue in the body, are particularly at risk of a thoracic aortic aneurysm. Those with Marfan syndrome may have a weakness in the aortic wall that makes them more susceptible to aneurysm. People with Marfan syndrome often have distinct physical traits, including tall stature, very long arms, a deformed breastbone and eye problems.Besides Marfan syndrome, other related disorders, such as Ehlers-Danlos and Loeys-Dietz syndromes, can contribute to a thoracic aortic aneurysm. Ehlers-Danlos syndrome causes your skin, joints and connective tissue to be fragile and makes your skin stretch easily.
  • Other medical conditions. Inflammatory conditions, such as giant cell arteritis and Takayasu arteritis, may cause thoracic aortic aneurysms.
  • Problems with your heart's aortic valve. Sometimes people who have problems with the valve that blood flows through as it leaves your heart (aortic valve) have an increased risk of thoracic aortic aneurysm. This is mainly true for people who were born with a bicuspid aortic valve, meaning the aortic valve has only two leaflets instead of three.
  • Untreated infection. Though it is a rare cause of thoracic aortic aneurysm, it's possible to develop this condition if you've had an untreated infection, such as syphilis or salmonella.
  • Traumatic injury. Rarely, some people who are injured in falls or motor vehicle crashes develop thoracic aortic aneurysms.

Risk Factors

Thoracic aortic aneurysm risk factors include:
  • Age. Thoracic aortic aneurysms occur most often in people age 65 and older.
  • Tobacco use. Tobacco use is a strong risk factor for the development of an aortic aneurysm.
  • High blood pressure. Increased blood pressure damages the blood vessels in the body, raising your chances of developing an aneurysm.
  • Buildup of plaques in your arteries (atherosclerosis). The buildup of fat and other substances that can damage the lining of a blood vessel (atherosclerosis) increases your risk of an aneurysm. This is a more common risk in older people.
  • Family history. People who have a family history of aortic aneurysm are at increased risk of having one. People who have a family history of aneurysms tend to develop aneurysms at a younger age and are at higher risk of rupture. This is a primary risk factor in younger people.
  • Marfan syndrome and related disorders. If you have Marfan syndrome or related disorders such as Loeys-Dietz syndrome or Ehlers-Danlos syndrome, you have a significantly higher risk of a thoracic aortic aneurysm.

Diagnosis

  • Chest X-ray
  • Echocardiogram
  • CT scan of chest
  • Magnetic Resonance Angiography (MRA)

Treatments

The size of the aneurysm, presence and severity of symptoms, and the risk of surgery help determine the treatment approach.

ccrn aortic aneurysm

REGULAR MONITORING is standard treatment for smaller aneurysms that do not require surgery. Plan on visiting your vascular surgeon regularly—usually once a year or every 6 months, depending on the size of the aneurysm—for a computed tomography (CT) scan or MRI to check the status and growth of the aneurysm. CONVENTIONAL SURGERY, a procedure called open thoracic aortic aneurysm repair or TAA, is done under a general anesthetic.
  • Through an incision along the side of the chest, a vascular surgeon uses special surgical tools to stop blood flow in the aorta above and below the aneurysm.
  • The section of the aorta with the aneurysm is replaced with an artificial graft.
  • The graft is sewn in place with fine stitches, and the incision is closed.
  • Most patients spend some time in the intensive care unit after surgery, and stay in the hospital 7-10 days.
ENDOVASCULAR TREATMENT is sometimes used and represents a less invasive approach. Thoracic aortic endograft repair (TEVAR) treats the aneurysm with a small device placed inside the aorta through a small incision or through puncture in the groin.

 

Critical Care Courses

Overview

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CCRN Predictor Exam

CCRN Predictor Exam

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CCRN Question Bank

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CCRN Review Course 

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CCRN Online Review 

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PCCN DKA Overview

PCCN DKA

PCCN DKA Overview

Pathophysiology

Diabetic ketoacidosis (DKA) is a serious complication of diabetes that occurs when your body produces high levels of blood acids called ketones.  DKA happens predominantly in those with Type 1 diabetes, but it can occur in those with Type 2 diabetes under certain circumstances.  This condition develops when you body can't produce enough insulin.  Insulin normally plays a key role in helping sugar (glucose) - a major source of energy for your muscles and other tissues - enter your cells.  Without enough insulin, your body begins to break down fat as fuel.  This process produces a buildup of acids in the bloodstream called ketones, eventually leading to diabetic ketoacidosis if untreated.   DKA is medical emergency, and without treatment it can lead to death.

Signs & Symptoms of DKA

DKA s/s often develop quickly, sometimes within 24 hours.  For some, these s/s may be the first indication of having diabetes.
  • Excessive thirst
  • Frequent urination
  • Nausea and vomiting
  • Abdominal pain
  • Weakness or fatigue
  • Fruity scented breath
  • Confusion

Causes of DKA

Sugar is a main source of energy for the cells that make up your muscles and other tissues.  Normally, insulin helps sugar enter your cells.  Without enough insulin, your body can't use sugar properly for energy.  This prompts the release of hormones that break down fat as fuel, which produces acids known as ketones.  Excess ketones build up in the blood and eventually spill over into the urine.
  • DKA is usually triggered by
  • An illness - an infection or other illness can cause your body to produce higher levels of certain hormones, such as adrenaline or cortisol.
  • A problem with insulin therapy - missed insulin treatments or inadequate insulin therapy can leave you with too little insulin in your system, triggering DKA.
  • Physical or emotional trauma
  • Heart attack
  • Alcohol or drug abuse, particularly cocaine
  • Corticosteroids and some diuretics

Risk Factors of DKA

  • Have Type 1 diabetes
  • Frequently miss insulin doses
  • May occur in Type 2 diabetes (uncommon)

Complications

DKA is treated with fluids, electrolytes such as sodium, potassium, and chloride and insulin.  Perhaps surprisingly, the most common complications of DKA are related to this lifesaving treatments. Treatment complications include
  • Low blood sugar (hypoglycemia)
  • Low potassium (hypokalemia)
  • Swelling in the brain (cerebral edema)

Treatment of DKA

  • Fluid replacement
  • Electrolyte replacement
  • Insulin therapy

Prevention of DKA

  • Commit to managing your diabetes
  • Monitor your blood sugar level
  • Adjust your insulin dosage as needed
  • Check your ketone levels
 

PCCN National Exam Courses

Overview

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How The Course Works

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PCCN Predictor Exam

PCCN Predictor Exam

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PCCN Question Bank

PCCN Question Bank

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PCCN Practice Questions

PCCN Practice Questions Bundle

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PCCN Review

PCCN Review Course 

  • Option 1
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  • Approved For 35 CEU's
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PCCN Online Review

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PCCN Online Review

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